Our Breeder Cardigan Corgi's

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All our puppies will be Akc Registered unless otherwise stated. They will have their 1st puppy shots, as well as multiple worming's before going to their new furever home's. 

Cardigan Welsh Corgi's all have long tails. 

All puppies will be given a 5 yr health guarantee as stated in the puppy contract. Meaning we guarantee they are free from any genetic health problems for up to 5 yr from purchase if kept in compliance as stated in the puppy contract.

 
Genetic Health Testing has been done on all of our adult dogs for: DM, EIC,  Pra-Rcd3, & VWD1. (scroll down to read about each of these genetic issues Cardigan Corgi's could have).

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Degenerative Myelopathy caused by Mutation of the SOD1 gene is an inherited neurologic disorder of dogs. This mutation is found in many breeds of dog, including the Pembroke Welsh corgi. While it is not clear for some of the other breeds, Pembroke Welsh corgis are known to develop degenerative myelopathy associated with this mutation. The variable presentation between breeds suggests that there are environmental or other genetic factors responsible for modifying disease expression. The disease affects the White Matter tissue of the spinal cord and is considered the canine equivalent to amyotrophic lateral sclerosis (Lou Gehrig’s disease) found in humans. Affected dogs usually present in adulthood with gradual muscle Atrophy and loss of coordination typically beginning in the hind limbs due to degeneration of the nerves. The condition is not typically painful for the dog, but will progress until the dog is no longer able to walk. The gait of dogs affected with degenerative myelopathy can be difficult to distinguish from the gait of dogs with hip dysplasia, arthritis of other joints of the hind limbs, or intervertebral disc disease. Late in the progression of disease, dogs may lose fecal and urinary continence and the forelimbs may be affected. Affected dogs may fully lose the ability to walk 6 months to 2 years after the onset of symptoms. Affected small breed dogs, such as the Pembroke Welsh corgi, often progress more slowly than affected large breed dogs and owners may postpone euthanasia until the dog is paraplegic.  

Breed-Specific Information for the Pembroke Welsh Corgi  

The Mutation of the SOD1 gene associated with degenerative myelopathy has been identified in the Pembroke Welsh corgi. The average age of onset for Pembroke Welsh corgis with degenerative myelopathy is approximately 11 years of age. The overall frequency of this disease is unreported in Pembroke Welsh corgis. However, in one study of 3209 Pembroke Welsh corgis tested, 28% were carriers of the mutation and 65.1% were at-risk/affected.  

Testing Tips: Genetic testing of the SOD1 gene in Pembroke Welsh corgis will reliably determine whether a dog is a genetic Carrier of degenerative myelopathy. Degenerative Myelopathy is inherited in an Autosomal Recessive manner in dogs meaning that they must receive two copies of the mutated gene (one from each parent) to develop the disease. In general, carrier dogs do not have features of the disease but when bred with another carrier of the same Mutation, there is a risk of having affected pups. Each pup that is born to this pairing has a 25% chance of inheriting the disease and a 50% chance of inheriting one copy and being a carrier of the SOD1 gene mutation. Reliable genetic testing is important for determining breeding practices. Because symptoms may not appear until adulthood and some at-risk/affected dogs do not develop the disease, genetic testing should be performed before breeding. Until the exact modifying environmental or genetic factor is determined, genetic testing remains the only reliable way to detect neurological disease associated with this mutation prior to death. In order to eliminate this mutation from breeding lines and to avoid the potential of producing affected pups, breeding of known carriers to each other is not recommended. Pembroke Welsh corgis that are not carriers of the mutation have no increased risk of having affected pups.  

Information copied from Paw Print Genetics

Von Willebrand Disease 1 (VWD1) is an inherited bleeding disorder affecting dogs. Dogs affected with VWDI have less than half of the normal level of Von Willebrand coagulation factor (vWf), which is an essential protein needed for normal blood clotting. There is variability in the amount of vWf such that not all dogs with two copies of the mutation are equally affected. Dogs that have less than 35% of the normal amount of vWf generally have mild to moderate signs of a bleeding disorder. Affected dogs may bruise easily, have frequent nosebleeds, bleed from the mouth when juvenile teeth are lost, and experience prolonged bleeding after surgery or trauma. Less often, the bleeding may be severe enough to cause death. Due to the variable severity of the disorder, affected dogs may not be identified until a surgery is performed or trauma occurs at which time excessive bleeding is noted. Veterinarians performing surgery on known affected dogs should have ready access to blood banked for transfusions. Most dogs will have a normal lifespan with this condition despite increased blood clotting times.

The Cardigan Welsh Corgi is included as a breed susceptible to Von Willebrand Disease 1 because of its close relatedness to the Pembroke Welsh Corgi which is known to carry the mutation of the VWF gene associated with Von Willebrand Disease 1. The frequency of the causal mutation in the general Cardigan Welsh Corgi population is unknown.

Information copied from Paw Print Genetics

As a quick refresher, recessive inherited diseases are those in which an individual must inherit two copies of a mutated gene (one from each parent) in order to develop the associated condition. Dogs inheriting two copies of the mutation are typically not recommended for breeding because even if bred to a dog that does not have the same genetic mutation, every puppy from the litter would inherit a single copy of the disease-associated mutation, thereby increasing the frequency of the mutation in the breed population to a significant degree. Dogs inheriting one copy of the mutation from a single parent are considered “carriers” of the disease and will not develop clinical signs of the disease themselves. However, when bred with another dog which carries the same mutation, approximately 25% of the offspring will inherit two copies of the mutation and will be at risk for or affected with the associated condition. Therefore, in general, it is recommended to only breed a carrier to a dog which did not inherit the same mutation. Using statistics as a guide, this strategy is expected to result in a litter consisting of approximately 50% carrier offspring and 50% normal or “clear” offspring. Thus, avoiding the removal of the carrier dog and its unique combination of genetic variants that contribute to the overall genetic diversity of the breed while limiting the number of puppies born with the known, disease-associated mutation.